During percutaneous renal tract creation in normally fixed kidneys, the puncture needle, dilators and sheath enter the renal parenchyma smoothly. However, when the kidney is abnormally mobile, it is pushed by the puncture needle and particularly by the dilators. Thus, when the dilators meet the resistance of the renal capsule, insisting to enter the parenchyma will lengthen the percutaneous tract [1]. This causes wide kinking of the guidewire, making dilator progression difficult [2]. Consequently, tract dilatation becomes time consuming with increased radiation exposure. To overcome this problem, we use a complement to ‘one-shot’ dilatation [3] by using bi-prong forceps to perform dissection of the renal parenchyma under direct vision and without radiation exposure